Index: qipapers.html
===================================================================
--- qipapers.html	(revision 556)
+++ qipapers.html	(revision 557)
@@ -14,5 +14,5 @@
      (see http://www.lri.fr/~filliatr/bibtex2html/),
      with the following command:
-     /usr/bin/bibtex2html -a -o - -t qipapers.html -s plain /tmp/tmpXHMMeN.bib  -->
+     /usr/bin/bibtex2html -a -o - -t qipapers.html -s plain /tmp/tmpOJrSp4.bib  -->
 
 <h1>qipapers.html</h1>
@@ -22468,7 +22468,7 @@
 C.M. Niessen, L.M.H. van&nbsp;der Raaij-Helmer, E.H.M. Hulsman, R.&nbsp;van&nbsp;der Neut,
   M.F. Jonkman, and A.&nbsp;Sonnenberg.
- Deficiency of the b4 subunit in junctional epidermolysis bullosa with
-  pyloric atresia: consequences for hemidesmosome formation and adhesion
-  properties.
+ Deficiency of the &beta;4 subunit in junctional epidermolysis
+  bullosa with pyloric atresia: consequences for hemidesmosome formation and
+  adhesion properties.
  <em>Journal of Cell Science</em>, 109:1695-1706, July 1996.
 
@@ -26934,7 +26934,8 @@
   Nievers, C.M. Niessen, R.D.M. Steenbergen, P.J.F. Snijders, and
   A.&nbsp;Sonnenberg.
- Hemidesmosome formation is initiated by the b4 integrin subunit,
-  requires complex formation of b4 and h.d1/plectin, and involves a direct
-  interaction between b4 and the bullous pemphigoid antigen.
+ Hemidesmosome formation is initiated by the &beta;4 integrin
+  subunit, requires complex formation of &beta;4 and h.d1/plectin, and
+  involves a direct interaction between &beta;4 and the bullous pemphigoid
+  antigen.
  <em>Journal of Cell Biology</em>, 142(1):271-284, July 1998.